Oxidative Stress and Neuroprotection
(Sprache: Englisch)
This book deals with basic and clinical aspects of monoamine oxidase (MAO) subtypes A and B highlighting its importance in neurological and psychiatric diseases. Consequently the therapeutic actions of MAO-A and -B inhibitors in Parkinson's disease (PK) and...
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Klappentext zu „Oxidative Stress and Neuroprotection “
This book deals with basic and clinical aspects of monoamine oxidase (MAO) subtypes A and B highlighting its importance in neurological and psychiatric diseases. Consequently the therapeutic actions of MAO-A and -B inhibitors in Parkinson's disease (PK) and depression are the focus of several chapters. As MAO is the basis of the development of the "oxidative stress hypothesis" of PD, several chapters are devoted to iron and iron-induced oxidative stress in various experimental studies and clinical conditions. Based on these findings, new compounds have been developed which not only block MAO, but are in addition, either inhibitors of acetylcholine esterase or have iron chelating properties. The aspect of "preclinical" and "clinical" neuro protection as well as MAO neuroprotection are additional topics covered in this book. MAO, iron and neuroprotection are seen in the framework of general anti Parkinson's therapy with chapters on levodopa, dopaminergic receptor agonists and clinical issues.Inhaltsverzeichnis zu „Oxidative Stress and Neuroprotection “
- Metal Specificity of an iron-responsive element in Alzheimer's APP mRNA 5' untranslated region, tolerance of SH-SY5Y and H4 neural cells to desferrioxamine, clioquinol, VK-28, and a piperazin chelator- Marker for a preclinical diagnosis of Parkinson's disease as a basis for neuroprotection
- Assessing neuroprotection in Parkinson's disease: from the animal models to molecular neuroimaging in vivo
- Acute and chronic affects of developmental iron deficiency on mRNA expression patterns in the brain
- A new look at levodopa based on the ELLDOPA study
- Altered regulation of iron transport and storage in Parkinson's disease
- The relationship of early studies of monoamine oxidase to present concepts
- Neuroprotection for Parkinson's disease
- Deprenyl: from chemical synthesis to neuroprotection
- Green tea catechins as brain-permeable, non toxic iron chelators to "iron out iron" from the brain
- Isatin interaction with glyceraldehyde-3-phosphate dehydrogenase, a putative target of neuroprotective drugs: partial agonism with deprenyl
- Isatin, an endogenous MAO inhibitor, and a rat model of Parkinson's disease induced by the Japanese encephalitis virus
- The DONPAD-study - treatment of dementia in patients with Parkinson's disease with donepezil
- Molecular mechanism of the relation of monoamine oxidase B and its inhibitors to Parkinson's disease: possible implications of glial cells
- Involvement of type A monoamine oxidase in neurodegeneration: regulation of mitochondrial signaling to cell death or neuroprotection
- Inhibition of amine oxidises by the histamine-1 receptor antagonist hydroxyzine
- Impact of selenium, iron, copper and zinc in on/off Parkinson's patients on L-dopa therapy
- Changing dopamine agonist treatment in Parkinson's disease: experiences with switching to pramipexole
- The use of rasagiline in Parkinson's disease
- Apoptosis inhibition in T cells triggers the expression of proinflammatory
... mehr
cytokines - implications for the CNS
- Moussa Youdim - an appreciation
- PD-related psychosis: pathophysiology with therapeutical strategies
- Long lasting effects of infancy iron deficiency - preliminary results
- Novel neuroprotective neurotrophic NAP analogs targeting metal toxicity and oxidative stress: potential candidates for the control of neurodegenerative diseases
- Moussa Youdim - an appreciation
- PD-related psychosis: pathophysiology with therapeutical strategies
- Long lasting effects of infancy iron deficiency - preliminary results
- Novel neuroprotective neurotrophic NAP analogs targeting metal toxicity and oxidative stress: potential candidates for the control of neurodegenerative diseases
... weniger
Bibliographische Angaben
- 2006, 2006, 264 Seiten, Masse: 21,5 x 28,3 cm, Gebunden, Englisch
- Herausgegeben von Parvez, S. H.; Riederer, Peter
- Herausgegeben: Peter Riederer, S. H. Parvez
- Verlag: Springer
- ISBN-10: 3211333274
- ISBN-13: 9783211333273
- Erscheinungsdatum: 15.09.2006
Sprache:
Englisch
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